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Fig. 1 | Diabetology & Metabolic Syndrome

Fig. 1

From: Proteomics of high-density lipoprotein subfractions and subclinical atherosclerosis in type 1 diabetes mellitus: a case–control study

Fig. 1

Differences in HDL2 and HDL3 protein abundance between T1D and controls. Volcano Plot indicating differences of protein abundance (log2 fold change) detected in HDL2 (A) and HDL3 (D) (significance in −log10) and odds ratio with 95% confidence interval for the association of HDL2 and HDL3 proteomics in T1D (B for HDL2 and E for HDL3) and controls (C for HDL2 and F for HDL3). Proteins are indicated by gene name abbreviation. AHSG alpha-2-Heremans-Schmid glycoprotein or fetuin-A, ALB albumin, APMAP adipocyte plasma membrane-associated protein, apoH apolipoprotein H or beta 2 glycoprotein 1, A1BG glycoprotein alpha-1B, CETP cholesteryl ester transfer protein, CST3 cystatin C, GC vitamin D binding protein or group-specific component, GPLD1 phosphatidylinositol-glycan-specific phospholipase D, HBB hemoglobin subunit beta, HPHPR haptoglobin or haptoglobin related protein, LCAT lecithin cholesterol acyltransferase, n.s. not significant, PCSK9 proprotein convertase subtilisin/kexin type 9, PLTP phospholipid transfer protein, PON1 paraoxonase-1, PON3 paraoxonase-3, RBP4 retinol binding protein type 4, SAA1 serum amyloid A1, SAA4 serum amyloid A4, TF Transferrin, TTR transthyretin

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