Substituting device-measured sedentary time with alternative 24-hour movement behaviours: compositional associations with adiposity and cardiometabolic risk in the ORISCAV-LUX 2 study

Table 5 Five-part compositional time-use: associations with adiposity and cardiometabolic risk markers per 30 min of prolonged sedentary time exchanged with non-prolonged sedentary time

	Prolonged → Non-prolonged sedentary time
	Model A	Model B	Model C
Waist circumference (cm)	− 0.29 (− 0.60 to 0.02)	− 0.30 (− 0.61 to 0.01)	–
Fasting glucose (mg/dL)	0.04 (− 0.29 to 0.37)	0.03 (− 0.30 to 0.36)	0.10 (− 0.22 to 0.43)
Triglycerides (%)	− 0.45 (− 1.58 to 0.69)	− 0.50 (− 1.63 to 0.64)	− 0.19 (− 1.28 to 0.91)
HDL-c (mg/dL)	0.13 (− 0.21 to 0.46)	0.14 (− 0.19 to 0.47)	0.04 (− 0.28 to 0.36)
CCMR (z-score)	− 0.03 (− 0.11 to 0.04)	− 0.04 (− 0.11 to 0.03)	–
Total body fat (%)	− 0.14 (− 0.32 to 0.03)	− 0.15 (− 0.32 to 0.03)	–
Fasting insulin (%)	− 0.90 (− 2.24 to 0.46)	− 0.90 (− 2.24 to 0.46)	− 0.23 (− 1.39 to 0.95)
ApoB/A1 ratio	0.00 (− 0.00 to 0.01)	0.00 (− 0.00 to 0.01)	0.00 (− 0.00 to 0.01)

Model A adjusted for sex, age, education level, season of measurement, smoking status, general health, medication use, and family history of cardiovascular disease or diabetes. Model B further adjusted for dietary factors (total caloric intake, salt and caffeine consumption), depressive symptoms, and sleep quality. Model C further adjusted for waist circumference. The results are β-coefficients (95% confidence intervals) and represent the expected difference in outcomes when replacing 30 min of prolonged with non-prolonged sedentary time, keeping all other movement behaviours constant at the compositional mean. Data for triglycerides and insulin were on the log scale, the results have been retransformed [(expβ-1)*100]–represent the percentage difference in outcomes per 30 min of time exchanged.
ApoB/A1 Apolipoprotein B–A1, CCMR Clustered cardiometabolic risk, HDL-c High-density lipoprotein cholesterol

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