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Fig. 2 | Diabetology & Metabolic Syndrome

Fig. 2

From: Association between osteocalcin, a pivotal marker of bone metabolism, and secretory function of islet beta cells and alpha cells in Chinese patients with type 2 diabetes mellitus: an observational study

Fig. 2Fig. 2

Comparison of glucose metabolism and islet alpha cell and beta cell secretory function among tertiles of OCN levels. PBG2h was lower in T3 group than group T2 and group T1 (15.02 ± 3.82 vs. 16.71 ± 5.36 vs. 16.64 ± 4.05, p < 0.05) (A). AUCins3h in T3 Group was significantly lower than T1 Group (53.45 (29.05, 93.65) vs. 78.48 (44.12, 123.72), p < 0.05) (B). Δcp0.5 h in T3 Group was significantly higher than T1Group and T2Group (0.53 (0.14, 1.06) vs. 0.36 (− 0.01, 0.80) vs. 0.24 (0.06, 0.62), p < 0.05) (C). GLA0.5 h in T3 group was lower than T2 and T1 groups (35.38 ± 17.90 vs. 41.51 ± 15.82 vs. 43.76 ± 19.09, p < 0.05) (D). Compared with the T1 group, GLA1h in the T3 group was lower (32.12 ± 12.80 vs. 38.32 ± 17.03, p < 0.05) (E). OCN Osteocalcin, PBG2h postprandial 2 h blood glucose, GLA0.5h postprandial 0.5 h glucagon, GLA1h postprandial 1 h glucagon, AUCins3h area under the curve of insulin, Δcp0.5h early postprandial secretion function of islet β cells

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