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Table 1 Baseline clinical and biochemical characterization of the study population

From: High exposure to phthalates is associated with HbA1c worsening in type 2 diabetes subjects with and without edentulism: a prospective pilot study

  DnE DE p
Age
(years)
67 ± 6 68 ± 7 ns
Male
(%)
75 71 ns
BMI
(Kg/m2)
30 ± 6 29 ± 4 ns
Alcohol intake low/medium/high
(%)
50/38/12 54/38/8 ns
T2D duration
(years)
7 [4; 10.5] 7.5 [4; 12] ns
Oral antidiabetic drugs
(%)
96 100 ns
Insulin
(%)
17 25 ns
Systolic blood pressure
(mmHg)
140 [131; 153] 145 [136; 159] ns
Diastolic blood pressure
(mmHg)
83 [73; 90] 82 [73; 90] ns
Fasting glucose
(mmol/L)
7.6 [6.0; 9.1] 7.8 [6.1; 10.7] ns
HbA1c
(mmol/mol)
52 [45; 56] 51 [44; 61] ns
eGFR
(ml/min/1·73m2)
85 [77; 92] 87 [73; 98] ns
CV events
(%)
17 17 ns
Total cholesterol
(mmol/L)
4.3 [3.9; 5.2] 4.3 [3.7; 5.1] ns
HDL cholesterol
(mmol/L)
1.1 [1.0; 1.4] 1.1 [1.0; 1.3] ns
LDL cholesterol
(mmol/L)
2.4 [1.8; 2.6] 2.5 [1.7; 3.3] ns
Triglycerides
(mmol/L)
1.7 [1.3; 2.3] 1.5 [1.1; 1.8] ns
ΣDEHP
(μg/g creatinine)
21.0 [13.3; 31.0] 22.6 [14.9; 40.0] ns
MEHP
(μg/g creatinine)
3.6 [2.2; 4.9] 3.5 [2.2; 5.2] ns
MEOHP
(μg/g creatinine)
4.8 [2.7; 6.8] 5.0 [3.5; 8.5] ns
MEHHP
(μg/g creatinine)
13.6 [6.8; 18.5] 13.8 [8.2; 24.6] ns
RMR1 4.8 [2.5; 5.5] 4.4 [2.5; 7.0] ns
RMR2 0.4 [0.3; 0.4] 0.4 [0.3; 0.5] ns
  1. RMR1 represents the rate of MEHP hydroxylation to MEHHP. RMR2 represents the rate of MEHHP oxidation to MEOHP