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Table 1 Demographic and biochemical characteristic in diabetic and non- diabetic patients with (+Ve) H. pylori and (−Ve) H. pylori infection

From: Oxidative DNA damage and oxidized low density lipoprotein in Type II diabetes mellitus among patients with Helicobacter pylori infection

Mean ± SD

 

Healthy (n = 50)

Non-diabetic (n = 50)

Diabetic (n = 50)

P value*

−ve H. pylori

−ve H. Pylori (n = 21)

+ve H. Pylori (n = 29)b

−ve H. Pylori (n = 17)

+ve H. Pylori (n = 33)b

M/F

41/9

7/10

3/30

12/9

19/10

<0.05*

Age (years)

27.18 ± 4.39

41.14 ± 7.95

44.86 ± 10.08

49.59 ± 8.47

51.79 ± 6.95

<0.0001*

BMI (Kg/m2)

25.52 ± 1.11

25.86 ± 1.68

27.72 ± 2.22

28.53 ± 2.24

28.88 ± 2.68

>0.05

F.B.S (mg/dl)a

78.64 ± 5.15

81.81 ± 7.08

82.79 ± 8.00

187.41 ± 57.32

182.91 ± 57.55

<0.0001*

2 h.PP.B.S (mg/dl)a

93.14 ± 6.99

101.67 ± 7.96

101.10 ± 12.39

341.76 ± 67.14

321.06 ± 79.81

<0.0001*

HbA1C %a

3.39 ± 0.49

3.84 ± 0.70

3.97 ± 0.52

7.30 ± 0.55

6.93 ± 0.69

<0.001*

H. Pylori IgG

0.86 ± 0.52

3.23 ± 1.73

66.01 ± 17.36

2.46 ± 2.20

94.52 ± 45.9

<0.001*

  1. * p < 0.05 is considered significant
  2. a References values: fasting blood glucose (F.B.S) 80–120 mg/dl; 2-hours postprandial blood glucose (2 h.pp.B.S)110–140 mg/dl; hemoglobin A1c (HbA1c) 3–6.5 %
  3. b The prevalence of H. pylori infection among diabetic and non-diabetics patients was 66 and 58 % (P = 0.001). Positive (+Ve) H. pylori infection and negative (−Ve) H. pylori infection