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Table 1 Demographic and biochemical characteristic in diabetic and non- diabetic patients with (+Ve) H. pylori and (−Ve) H. pylori infection

From: Oxidative DNA damage and oxidized low density lipoprotein in Type II diabetes mellitus among patients with Helicobacter pylori infection

Mean ± SD
  Healthy (n = 50) Non-diabetic (n = 50) Diabetic (n = 50) P value*
−ve H. pylori −ve H. Pylori (n = 21) +ve H. Pylori (n = 29)b −ve H. Pylori (n = 17) +ve H. Pylori (n = 33)b
M/F 41/9 7/10 3/30 12/9 19/10 <0.05*
Age (years) 27.18 ± 4.39 41.14 ± 7.95 44.86 ± 10.08 49.59 ± 8.47 51.79 ± 6.95 <0.0001*
BMI (Kg/m2) 25.52 ± 1.11 25.86 ± 1.68 27.72 ± 2.22 28.53 ± 2.24 28.88 ± 2.68 >0.05
F.B.S (mg/dl)a 78.64 ± 5.15 81.81 ± 7.08 82.79 ± 8.00 187.41 ± 57.32 182.91 ± 57.55 <0.0001*
2 h.PP.B.S (mg/dl)a 93.14 ± 6.99 101.67 ± 7.96 101.10 ± 12.39 341.76 ± 67.14 321.06 ± 79.81 <0.0001*
HbA1C %a 3.39 ± 0.49 3.84 ± 0.70 3.97 ± 0.52 7.30 ± 0.55 6.93 ± 0.69 <0.001*
H. Pylori IgG 0.86 ± 0.52 3.23 ± 1.73 66.01 ± 17.36 2.46 ± 2.20 94.52 ± 45.9 <0.001*
  1. * p < 0.05 is considered significant
  2. a References values: fasting blood glucose (F.B.S) 80–120 mg/dl; 2-hours postprandial blood glucose (2 h.pp.B.S)110–140 mg/dl; hemoglobin A1c (HbA1c) 3–6.5 %
  3. b The prevalence of H. pylori infection among diabetic and non-diabetics patients was 66 and 58 % (P = 0.001). Positive (+Ve) H. pylori infection and negative (−Ve) H. pylori infection
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